Mutated recombinant human glucagon-like peptide-1 protects SH-SY5Y cells from apoptosis induced by amyloid-beta peptide (1-42).
Accumulation and deposition of amyloid beta peptide (Abeta) within the mind causes neuronal apoptosis and finally results in Alzheimer’s illness (AD). A therapeutic goal for AD is to dam the cascade response induced by Abeta. It has been demonstrated that … Read More